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Cayman Chemical anti-mouse primary antibodies glial fibrillary acidic protein (gfap)
UDCA treatment halts reactive gliosis and preserves neuronal cells in OIR mice. Frozen retinal sections from control mice at P17 (room air), OIR mice, and OIR mice treated with 50 mg/kg UDCA (P7–P17) were stained with ( A ) <t>GFAP</t> antibody. Total protein fractions from control mice (room air), OIR mice at P17, and OIR mice treated with 50 mg/kg UDCA (P7–P17) were used to analyze ( B ) protein levels of GFAP by Western blotting. ( C ) Densitometry graphs showing changes in glial <t>fibrillary</t> acidic protein (GFAP) protein expressions. Values are mean ± S.D. ( n = 6 retinas per group). ( D , E ) Frozen retinal sections from control mice at P17 (room air), OIR mice, and OIR mice treated with 50 mg/kg UDCA (P7–P17) were stained with RNA binding protein with multiple splicing (RBPMS) antibody and positive cells were counted. RBPMS data are expressed as cell number/100 μm retinal length and presented as a percent change from control. * p < 0.05 vs. control and # p < 0.05 vs. OIR. GLC: ganglion cell layer; IPL: inner plexiform layer; INL: inner nuclear layer; ONL: outer nuclear layer; RPE: retinal pigmented epithelium; RBPMS: RNA Binding Protein with Multiple Splicing; GFAP: Glial fibrillary acidic protein.
Anti Mouse Primary Antibodies Glial Fibrillary Acidic Protein (Gfap), supplied by Cayman Chemical, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
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Interchim Chemicals primary antibody mouse anti-glial fibrillary acidic protein (gfap) ayz280
UDCA treatment halts reactive gliosis and preserves neuronal cells in OIR mice. Frozen retinal sections from control mice at P17 (room air), OIR mice, and OIR mice treated with 50 mg/kg UDCA (P7–P17) were stained with ( A ) <t>GFAP</t> antibody. Total protein fractions from control mice (room air), OIR mice at P17, and OIR mice treated with 50 mg/kg UDCA (P7–P17) were used to analyze ( B ) protein levels of GFAP by Western blotting. ( C ) Densitometry graphs showing changes in glial <t>fibrillary</t> acidic protein (GFAP) protein expressions. Values are mean ± S.D. ( n = 6 retinas per group). ( D , E ) Frozen retinal sections from control mice at P17 (room air), OIR mice, and OIR mice treated with 50 mg/kg UDCA (P7–P17) were stained with RNA binding protein with multiple splicing (RBPMS) antibody and positive cells were counted. RBPMS data are expressed as cell number/100 μm retinal length and presented as a percent change from control. * p < 0.05 vs. control and # p < 0.05 vs. OIR. GLC: ganglion cell layer; IPL: inner plexiform layer; INL: inner nuclear layer; ONL: outer nuclear layer; RPE: retinal pigmented epithelium; RBPMS: RNA Binding Protein with Multiple Splicing; GFAP: Glial fibrillary acidic protein.
Primary Antibody Mouse Anti Glial Fibrillary Acidic Protein (Gfap) Ayz280, supplied by Interchim Chemicals, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
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Synaptic Systems primary monoclonal mouse anti-gfap antibody #173011
UDCA treatment halts reactive gliosis and preserves neuronal cells in OIR mice. Frozen retinal sections from control mice at P17 (room air), OIR mice, and OIR mice treated with 50 mg/kg UDCA (P7–P17) were stained with ( A ) <t>GFAP</t> antibody. Total protein fractions from control mice (room air), OIR mice at P17, and OIR mice treated with 50 mg/kg UDCA (P7–P17) were used to analyze ( B ) protein levels of GFAP by Western blotting. ( C ) Densitometry graphs showing changes in glial <t>fibrillary</t> acidic protein (GFAP) protein expressions. Values are mean ± S.D. ( n = 6 retinas per group). ( D , E ) Frozen retinal sections from control mice at P17 (room air), OIR mice, and OIR mice treated with 50 mg/kg UDCA (P7–P17) were stained with RNA binding protein with multiple splicing (RBPMS) antibody and positive cells were counted. RBPMS data are expressed as cell number/100 μm retinal length and presented as a percent change from control. * p < 0.05 vs. control and # p < 0.05 vs. OIR. GLC: ganglion cell layer; IPL: inner plexiform layer; INL: inner nuclear layer; ONL: outer nuclear layer; RPE: retinal pigmented epithelium; RBPMS: RNA Binding Protein with Multiple Splicing; GFAP: Glial fibrillary acidic protein.
Primary Monoclonal Mouse Anti Gfap Antibody #173011, supplied by Synaptic Systems, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
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Boehringer Mannheim mouse primary antibody against gfap boehringer mannheim frg
UDCA treatment halts reactive gliosis and preserves neuronal cells in OIR mice. Frozen retinal sections from control mice at P17 (room air), OIR mice, and OIR mice treated with 50 mg/kg UDCA (P7–P17) were stained with ( A ) <t>GFAP</t> antibody. Total protein fractions from control mice (room air), OIR mice at P17, and OIR mice treated with 50 mg/kg UDCA (P7–P17) were used to analyze ( B ) protein levels of GFAP by Western blotting. ( C ) Densitometry graphs showing changes in glial <t>fibrillary</t> acidic protein (GFAP) protein expressions. Values are mean ± S.D. ( n = 6 retinas per group). ( D , E ) Frozen retinal sections from control mice at P17 (room air), OIR mice, and OIR mice treated with 50 mg/kg UDCA (P7–P17) were stained with RNA binding protein with multiple splicing (RBPMS) antibody and positive cells were counted. RBPMS data are expressed as cell number/100 μm retinal length and presented as a percent change from control. * p < 0.05 vs. control and # p < 0.05 vs. OIR. GLC: ganglion cell layer; IPL: inner plexiform layer; INL: inner nuclear layer; ONL: outer nuclear layer; RPE: retinal pigmented epithelium; RBPMS: RNA Binding Protein with Multiple Splicing; GFAP: Glial fibrillary acidic protein.
Mouse Primary Antibody Against Gfap Boehringer Mannheim Frg, supplied by Boehringer Mannheim, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
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Covance tuji or gfap mouse polyclonal primary antibody
UDCA treatment halts reactive gliosis and preserves neuronal cells in OIR mice. Frozen retinal sections from control mice at P17 (room air), OIR mice, and OIR mice treated with 50 mg/kg UDCA (P7–P17) were stained with ( A ) <t>GFAP</t> antibody. Total protein fractions from control mice (room air), OIR mice at P17, and OIR mice treated with 50 mg/kg UDCA (P7–P17) were used to analyze ( B ) protein levels of GFAP by Western blotting. ( C ) Densitometry graphs showing changes in glial <t>fibrillary</t> acidic protein (GFAP) protein expressions. Values are mean ± S.D. ( n = 6 retinas per group). ( D , E ) Frozen retinal sections from control mice at P17 (room air), OIR mice, and OIR mice treated with 50 mg/kg UDCA (P7–P17) were stained with RNA binding protein with multiple splicing (RBPMS) antibody and positive cells were counted. RBPMS data are expressed as cell number/100 μm retinal length and presented as a percent change from control. * p < 0.05 vs. control and # p < 0.05 vs. OIR. GLC: ganglion cell layer; IPL: inner plexiform layer; INL: inner nuclear layer; ONL: outer nuclear layer; RPE: retinal pigmented epithelium; RBPMS: RNA Binding Protein with Multiple Splicing; GFAP: Glial fibrillary acidic protein.
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OriGene gfap mouse monoclonal antibody
UDCA treatment halts reactive gliosis and preserves neuronal cells in OIR mice. Frozen retinal sections from control mice at P17 (room air), OIR mice, and OIR mice treated with 50 mg/kg UDCA (P7–P17) were stained with ( A ) <t>GFAP</t> antibody. Total protein fractions from control mice (room air), OIR mice at P17, and OIR mice treated with 50 mg/kg UDCA (P7–P17) were used to analyze ( B ) protein levels of GFAP by Western blotting. ( C ) Densitometry graphs showing changes in glial <t>fibrillary</t> acidic protein (GFAP) protein expressions. Values are mean ± S.D. ( n = 6 retinas per group). ( D , E ) Frozen retinal sections from control mice at P17 (room air), OIR mice, and OIR mice treated with 50 mg/kg UDCA (P7–P17) were stained with RNA binding protein with multiple splicing (RBPMS) antibody and positive cells were counted. RBPMS data are expressed as cell number/100 μm retinal length and presented as a percent change from control. * p < 0.05 vs. control and # p < 0.05 vs. OIR. GLC: ganglion cell layer; IPL: inner plexiform layer; INL: inner nuclear layer; ONL: outer nuclear layer; RPE: retinal pigmented epithelium; RBPMS: RNA Binding Protein with Multiple Splicing; GFAP: Glial fibrillary acidic protein.
Gfap Mouse Monoclonal Antibody, supplied by OriGene, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
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UDCA treatment halts reactive gliosis and preserves neuronal cells in OIR mice. Frozen retinal sections from control mice at P17 (room air), OIR mice, and OIR mice treated with 50 mg/kg UDCA (P7–P17) were stained with ( A ) GFAP antibody. Total protein fractions from control mice (room air), OIR mice at P17, and OIR mice treated with 50 mg/kg UDCA (P7–P17) were used to analyze ( B ) protein levels of GFAP by Western blotting. ( C ) Densitometry graphs showing changes in glial fibrillary acidic protein (GFAP) protein expressions. Values are mean ± S.D. ( n = 6 retinas per group). ( D , E ) Frozen retinal sections from control mice at P17 (room air), OIR mice, and OIR mice treated with 50 mg/kg UDCA (P7–P17) were stained with RNA binding protein with multiple splicing (RBPMS) antibody and positive cells were counted. RBPMS data are expressed as cell number/100 μm retinal length and presented as a percent change from control. * p < 0.05 vs. control and # p < 0.05 vs. OIR. GLC: ganglion cell layer; IPL: inner plexiform layer; INL: inner nuclear layer; ONL: outer nuclear layer; RPE: retinal pigmented epithelium; RBPMS: RNA Binding Protein with Multiple Splicing; GFAP: Glial fibrillary acidic protein.

Journal: Journal of Clinical Medicine

Article Title: Ursodeoxycholic Acid Halts Pathological Neovascularization in a Mouse Model of Oxygen-Induced Retinopathy

doi: 10.3390/jcm9061921

Figure Lengend Snippet: UDCA treatment halts reactive gliosis and preserves neuronal cells in OIR mice. Frozen retinal sections from control mice at P17 (room air), OIR mice, and OIR mice treated with 50 mg/kg UDCA (P7–P17) were stained with ( A ) GFAP antibody. Total protein fractions from control mice (room air), OIR mice at P17, and OIR mice treated with 50 mg/kg UDCA (P7–P17) were used to analyze ( B ) protein levels of GFAP by Western blotting. ( C ) Densitometry graphs showing changes in glial fibrillary acidic protein (GFAP) protein expressions. Values are mean ± S.D. ( n = 6 retinas per group). ( D , E ) Frozen retinal sections from control mice at P17 (room air), OIR mice, and OIR mice treated with 50 mg/kg UDCA (P7–P17) were stained with RNA binding protein with multiple splicing (RBPMS) antibody and positive cells were counted. RBPMS data are expressed as cell number/100 μm retinal length and presented as a percent change from control. * p < 0.05 vs. control and # p < 0.05 vs. OIR. GLC: ganglion cell layer; IPL: inner plexiform layer; INL: inner nuclear layer; ONL: outer nuclear layer; RPE: retinal pigmented epithelium; RBPMS: RNA Binding Protein with Multiple Splicing; GFAP: Glial fibrillary acidic protein.

Article Snippet: Slides were fixed in 4% paraformaldehyde and incubated overnight at 4 °C with anti-mouse primary antibodies used at the following concentrations: 4-hydroxynonenal (4-HNE) (1:100; Abcam, Cambridge, MA, USA), glial fibrillary acidic protein (GFAP) (1:200; Cayman Chemical, Ann Arbor, MI, USA), and RNA binding protein with multiple splicing (RBPMS) (1:500; GeneTex, Alton Pkwy Irvine, CA, USA).

Techniques: Control, Staining, Western Blot, RNA Binding Assay